4/29/2023 0 Comments The scramblerHowever, the qualitative element is that these electric impulses are the basic information code by which the nervous system can be interpreted in a cybernetic model. In a nutshell, the gate control theory addresses the activity of the nervous system in terms of “quantitative” electric impulses. For this same reason, Scrambler Therapy requires careful positioning of the electrodes, always guided by patient feedback, so as to use only nerve pathways that do not present structural or functional alterations capable of degrading or not correctly conveying the synthetic information of “non-pain.” The result of the loss of information due to the impossibility of transmitting it correctly always transforms Scrambler Therapy emissions into simple electrical stimulus, which in addition to being ineffective, can easily produce pain instead of analgesia. In fact, if you eliminate the non-pain information from the emissions of Scrambler Therapy making it similar to a TENS (transcutaneous electrical nerve stimulation), this is exactly what happens. Therefore, if we consider only the differential effect of the electrical activities between these 2 branches as required by the gate control theory, the stimulation of the C fibers must produce pain. It is interesting to note that the gate control theory is apparently in complete contrast with the Scrambler Therapy model, since C fibers and not A-Beta fibers are stimulated. On the other hand, in acute pain, where the cause/effect relationship between nociceptive stimulus and lasting pain follows the normal physiological response, the gate control theory is consistent and is confirmed by experiments. Since chronic neuropathic pain is characterized by abnormal function of the somatosensory nervous system, the gate control theory does not easily lend itself to the development of a new type of therapy. However, it has been revised and updated and some aspects of the theory have been redefined. 1 Despite the time elapsed since its introduction in 1965, this theory still remains relevant today. A long-standing and commonly accepted model used to understand mechanisms of pain transmission and perception has been the gate control theory. Scrambler therapy is aimed at creating a non-invasive highly effective treatment for chronic neuropathic and cancer pain, which is resistant to other treatments. The aim of this review is to clarify the underlying theory of Scrambler Therapy and describe the appropriate usage method that maximizes its effectiveness while reducing bias and deepen the explanation of the artificial neuron technology associated with Scrambler Therapy. The goal of Scrambler Therapy is to eliminate pain during treatment and allow for long-lasting analgesia after a series of 10 to 12 consecutive treatments performed over a 2-week period. Scrambler therapy was developed to treat chronic neuropathic pain and cancer pain resistant to opioids and other types of treatments. This is a different theoretical mechanism than the traditional electric stimulation of A-Beta fibers to produce paresthesia and/or block the conduction of nerve fibers to produce an analgesic effect, that is, via TENS (transcutaneous electrical nerve stimulation) machines. The active principle with Scrambler Therapy is such that synthetic “non-pain” information is transmitted by C fiber surface receptors. Scrambler therapy (ST) is an electro-analgesia therapy for the noninvasive treatment of chronic neuropathic and cancer pain based on a new generation of medical device that uses 5 artificial neurons and is based on a novel theoretical model the differs from gate control theory.
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